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Not yet recruiting NCT06952140

Hemodynamic Effects of Ketone Esters in Patients With Sepsis Induced Cardiomyopathy

No phase Interventional Sepsis Induced Cardiomyopathy

For patients and families

In plain language

An automatic summary of structured registry data. It is an orientation aid, not a substitute for the official protocol or a physician assessment.

What is being studied
The protocol lists: Ketone ester, Placebo.
Who it may be relevant to
Registry conditions: Sepsis Induced Cardiomyopathy. Basic parameters: from 18 years · All.
What needs checking
Age, condition and sex are only basic indicators. Prior treatment, laboratory values and other mandatory requirements appear in the eligibility criteria below.
Where it takes place
Center list to be confirmed — check the primary protocol.
Next step
Save the trial, show it to the treating physician, and confirm current recruitment with the study center. Costs, documents and travel →

Overview

Sepsis is a life-threatening organ dysfunction caused by a dysregulated host response to infection and is associated with a high mortality rate in the ICU. Sepsis induced cardiomyopathy (SICM) is a multi-factorial process that appears in approximately 50% of patients with sepsis/septic shock and is associated with increased mortality. It is suggested that ketone bodies are more efficient substrates of energy metabolism than glucose, with a lower oxygen consumption per ATP-molecule produced and that the failing human heart increases the capacity to metabolize ketones. Previous studies have found acute beneficial hemodynamic effects of ketone esters in patients with chronic heart failure and cardiogenic shock, respectively. Improved hemodynamics and reduced systemic oxygen consumption as an effect of ketone esters might be of great benefit in patients admitted to the ICU. Thus, the investigators aim to investigate the hemodynamic effects of ketone esters in patients with sepsis induced cardiomyopathy in this randomized, placebo-controlled, double-blinded, cross-over, acute intervention study. .

Interventions

  • Dietary supplement Ketone ester
    Ketone ester: 3-hydroxybutyrate as enteral bolus (500 mg/kg)
  • Dietary supplement Placebo
    Maltodextrin (isovolumic and isocaloric placebo) as enteral bolus

Primary outcome measures

  • Global longitudinal strain [Time frame: From intervention to 3 hours after intervention (this is assessed for both treatment arms)]
Secondary outcome measures (7)
  • Left ventricular ejection fraction [Time frame: From intervention to 3 hours after intervention (this is assessed for both treatment arms)]
  • Mean arterial pressure [Time frame: From intervention to 3 hours after intervention (this is assessed for both treatment arms)]
  • Cardiac output [Time frame: From intervention to 3 hours after intervention (this is assessed for both treatment arms)]
  • Peripheral blood oxygen saturation [Time frame: From intervention to 3 hours after intervention (this is assessed for both treatment arms)]
  • Arterial blood pH [Time frame: From intervention to 3 hours after intervention (this is assessed for both treatment arms)]
  • Arterial blood lactate [Time frame: From intervention to 3 hours after intervention (this is assessed for both treatment arms)]
  • Accumulated norepinephrine [Time frame: From intervention to 3 hours after intervention (this is assessed for both treatment arms)]

Eligibility criteria

Inclusion criteria

  • Patients ≥ 18 years of age admitted to the the intensive care unit (ICU)
  • LVEF < 50% determined by a screening echocardiography and analysed according to the Simpson biplane method
  • Ability for study personnel to perform transthoracic echocardiography
  • Suspected or documented infection (suspected infection is defined as ongoing antibiotic treatment and/or body fluid culture sampling performed within 72 hours before screening)

Exclusion criteria

  • Diagnosis of heart failure with reduced ejection fraction prior to ICU admission according to health records
  • Surgical cause of ICU admission
  • For patients in shock: Other primary causes of shock than sepsis (i.e. hypovolemia, haemorrhage, cardiogenic etiology, pulmonary embolism, anaphylaxis)
  • Blood pH < 7.20
  • Severe gastroparesis
  • Inability to position a nasogastric tube

Criteria are shown verbatim from the registry (in English). Final eligibility is always assessed by the study center.

Healthy volunteers: No

Study design

Allocation
Randomized
Model
Crossover
Masking
Triple blind
Primary purpose
Treatment

Study locations

Center list to be confirmed — check the primary protocol.

Identifiers

NCT: NCT06952140 · KetoSIC

Primary sources (government registries)

View this study on ClinicalTrials.gov ↗